NURS 6501 Week 2 Module 1 Assignment Analyze a Case Study
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NURS 6501 Week 2 Module 1 Assignment Analyze a Case Study
Physiological Modifications
The signs and symptoms are a result of the infection with which the patient was diagnosed, which caused the patient to become septic. Increased blood flow to the area results in the redness seen in the wound. Due to the increased number of neutrophils, white blood cells, nutrient factors, and enzymes deposited in the infected area to aid in the fight against the infection, the area will swell (Hughes, 2016). Chemicals emitted by white blood cells, combined with increased activity at
NURS 6501 Week 2 Module 1 Assignment Analyze a Case Study
the site, may result in localized heat and tenderness. Interleukin 10 (IL 10) is a pro-inflammatory cytokine that protects the host and promotes normal tissue homeostasis. Additionally, it is known as Cytokine Synthesis Inhibitory Factor (CSIF).
NRAMP1 is primarily expressed in macrophages and monocytes and is involved in iron metabolism and host resistance to specific pathogens. It also regulates natural resistance to infection. IL 4 is a cytokine that induces young helper T cells to differentiate into Th2 cells (Cianfarani, Zambruno, Castiglia, & Odorisio, 2017).
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Interleukin 4 has anti-inflammatory properties and inhibits tumor necrosis factor production. Immunosuppression can result in an increased susceptibility to opportunistic infections due to an immune deficiency in the body, and even the tiniest pathogens can become pathogenic when the immune system is compromised (Larouche, Sheoran, Maruyama, & Martino, 2018). Immunosuppression may be induced in the body to decrease the risk of allograft rejection and increase the likelihood of survival following transplantation. Allograft vasculopathy may develop spontaneously.
References
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M. A. Hughes (March 24, 2016).
Wound infection: a knowledge gap that must be addressed
British Journal of Nursing, Volume 25, Number 6.)
DOI: 10.12968/bjon.2016.25.6.S46
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F. Cianfarani, G. Zambruno, D. Castiglia, et al (July 01, 2017).
Altered Wound Healing Pathophysiology in Recessive Dystrophic Epidermolysis Bullosa
American Journal of Pathology, vol. 187, no. 7, pp. 1445-1453.
https://doi.org/10.1016/j.ajpath.2017.03.003
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J. Larouche, S. Sheoran, K. Maruyama, and M. M. Martino (July 01, 2018).
Mechanisms and Novel Therapeutic Targets in Immune Regulation of Skin Wound Healing.
Advances in Wound Care, 7, no. 7 (2007), pp. 209-231.
https://doi.org/10.1089/wound.2017.0761
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